Spoilers for the upcoming Magic: The Gathering set Wilds of Eldraine begin next week.

Scientists have discovered a ghost-white shark with a rare condition. The predator was accidentally hooked by fishermen dedicated to catching sole. They were working from the coastal area of Los Chimus in Peru and informed authorities of the find. At the time of its capture, the shark had injuries to its gill slits as a result of being caught up in the net last month. The marine animal was transferred to the Peruvian Institute of the Sea (IMARPE) in Chimbote to undergo tests. Scientists discovered that the shark had leucism, which causes partial loss of pigmentation. The condition differs to albinism, which is a complete lack of melanin - a substance in the body that produces hair, eye and skin pigmentation. Albinos often have pink or red eyes but while leucism often makes an animal’s skin white, it does not affect their eye colour. IMARPE said this is the first known case of a shark in Peruvian waters having this condition. The fish was a young female that measured 89cm in length. Sexual maturity in females takes place when they are around 220cm in length. The predatory species sits high on the trophic food chain and mostly feeds on other sharks, rays, cetaceans and sea lions. Broadnose sevengill sharks are known to hunt in packs to bring down large prey. They are recognisable by their seven gill slits as most shark species have five. Sign up for our free Indy100 weekly newsletter How to join the indy100's free WhatsApp channel Have your say in our news democracy. Click the upvote icon at the top of the page to help raise this article through the indy100 rankings

TURIN, Italy--(BUSINESS WIRE)--Sep 4, 2023--

Growing up, people are generally taught that taking vitamins is good for you, with many people taking supplements to boost their intake. However, results of a new study suggest that taking vitamins could actually have a negative impact on health as they may help tumours to grow. New research suggests that taking additional vitamins and minerals, including common antioxidants such as vitamins A and C, and selenium and zinc, could help the blood vessels in tumours to grow. The study was conducted by scientists at the Karolinska Institutet in Sweden who published their findings in the Journal of Clinical Investigation. The results were somewhat of a surprise to experts, who believed anti-oxidants to be protective of the body. In their research, they found that vitamin C and other antioxidants actually stimulated the formation of new blood vessels in lung cancer tumours. Martin Bergö, who is a professor at the Department of Biosciences and Nutrition, said: “We’ve found that antioxidants activate a mechanism that causes cancer tumours to form new blood vessels, which is surprising since it was previously thought that antioxidants have a protective effect. “The new blood vessels nourish the tumours and can help them grow and spread.” Antioxidants, in normal amounts, neutralise free oxygen radicals that damage the body, so are beneficial. But, scientists found that high doses of antioxidants can activate a protein called BACH1 which begins the formation of new blood vessels, allowing harmful tumours to grow. Bergö explained: “There’s no need to fear antioxidants in normal food but most people don’t need additional amounts of them. “In fact, it can be harmful for cancer patients and people with an elevated cancer risk.” Ting Wang, the study’s lead author, said: “Many clinical trials have evaluated the efficacy of angiogenesis inhibitors, but the results have not been as successful as anticipated. “Our study opens the door to more effective ways of preventing angiogenesis in tumours; for example, patients whose tumours exhibit high levels of BACH1 might benefit more from anti-angiogenesis therapy than patients with low BACH1 levels.” Sign up to our free Indy100 weekly newsletter Have your say in our news democracy. Click the upvote icon at the top of the page to help raise this article through the indy100 rankings.

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MANILA, Philippines--(BUSINESS WIRE)--Jul 3, 2023--

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