A new lawsuit filed against Nvidia alleged that a file accidentally left on display by one of its employees during a video conference contains data stolen from the car tech company Valeo. The lawsuit alleged that employee Mohammad Moniruzzaman downloaded the “entirety of Valeo’s advanced parking and driving assistance systems source code” in early 2021 without authorization. It says he also downloaded “scores of Valeo Word documents, PowerPoint presentations, PDF files, and Excel spreadsheets explaining various aspects of the technology” before leaving to join Nvidia in August of that year. Valeo is accusing Nvidia of having benefited from the stolen trade secrets. “Mr Moniruzzaman ended his employment at Valeo and took the stolen source code and technical documentation with him to Nvidia, receiving a promotion to a senior position working on the software development for the very same project,” the lawsuit alleged. The alleged data theft reportedly came to light the following year when staff from both Nvidia and Valeo were working over a Microsoft Teams video conference call on a joint parking assistance project. Nvidia was developing the software, while Valeo was providing ultrasonic sensor hardware in the collaborative project for an unnamed automotive parts manufacturer. “On March 8, 2022, one of these videoconference meetings was scheduled. Mr Moniruzzaman, now employed by Nvidia, attended the video conference call… and shared his computer screen during the call,” the lawsuit noted. “When he minimized the PowerPoint presentation he had been sharing, however, he revealed one of Valeo’s verbatim source code files open on his computer,” it said. Valeo participants on the conference call recognised the source code and took a screenshot of it, the company said. “So brazen was Mr Moniruzzaman’s theft, the file path on his screen still read ‘ValeoDocs,’” the lawsuit noted. Mr Moniruzzaman had admitted to stealing Valeo’s software and using it while employed at Nvidia when he was questioned by German police, according to Valeo’s lawsuit. Nvidia has responded that it has no interest in using the stolen code, according to The Verge. But Valeo alleged that its competitor has benefited from the data, which it says would save “millions of dollars in development costs”. Citing these reasons, Valeo is seeking recovery of damages and an injunction to stop Nvidia and its staff from using its trade secrets. The Independent has reached out to Mr Moniruzzaman through Nvidia for a comment. Read More Trump’s Truth Social sues 20 media outlets over financial loss reports AI breakthrough could help us build solar panels out of ‘miracle material’ ‘We let you down’: Peloton apologises for Thanksgiving ride AI breakthrough could help us build solar panels out of ‘miracle material’ ‘We let you down’: Peloton apologises for Thanksgiving ride HSBC app not working on one of the biggest shopping days of the year

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Alcohol addiction ruins millions of lives every year, but scientists may have found a cure for this terrible affliction. A new treatment for alcohol use disorder (AUD) has been trialled in monkeys with impressive results and, if these translate to human trials, the impact could be monumental. A team of neuroscientists and physiologists from across the US tested a new type of gene therapy to see if they could directly target the underlying brain circuitry associated with sustained heavy drinking. As they noted, in the journal Nature Medicine, people suffering from AUD commonly return to alcohol use even if they attempt to quit. This is largely to do with what’s known as mesolimbic dopamine (DA) signalling – meaning how the central nervous system circuit communicates the feelgood neurotransmitter dopamine. A protein called glial-derived neurotrophic factor (GDNF) is key to keeping these neurons in this reward circuitry functioning. However, experts have found that levels of GDNF are reduced in people with AUD during periods of alcohol abstinence, most notably in a region of the brain called the ventral tegmental area (VTA), as IFLScience notes. Therefore, the researchers decided to test whether using gene therapy to deliver more GDNF to the VTA could help reinforce this crucial dopaminergic signalling and prevent patients from suffering an alcoholic relapse. The team of scientists explained how alcohol consumption in non-addicts prompts the release of dopamine, creating a pleasurable buzz feeling, but chronic alcohol use causes the brain to adapt and stop releasing so much dopamine. “So when people are addicted to alcohol, they don’t really feel more pleasure in drinking,” Dr Kathleen Grant, a senior co-author of the study, said in a statement. “It seems that they’re drinking more because they feel a need to maintain an intoxicated state.” For their research, Dr Grant and her colleagues used eight rhesus macaque monkeys, who were exposed to increasing concentrations of alcohol over four 30-day “induction” periods. The monkeys then had free access to alcohol and water for 21 hours a day for six months, during which they developed heavy drinking behaviours. This was then followed by a 12-week abstinence phase, with the GDNF treatment performed four weeks in for half of the subjects. The gene therapy was delivered using a a viral vector containing a copy of the human GDNF gene injected directly into the primate’s VTA, according to IFLScience. And the results were truly jaw-dropping. “Drinking went down to almost zero,” Dr Grant said. “For months on end, these animals would choose to drink water and just avoid drinking alcohol altogether. They decreased their drinking to the point that it was so low we didn’t record a blood-alcohol level.” The most exciting aspect of their findings is the suggestion that gene therapy could offer a permanent solution for people with the most severe cases of AUD. This will be a welcome glimmer of hope to many, given that some 29.5 million people were diagnosed with AUD in the US alone in 2021, according to the National Institute on Alcohol Abuse and Alcoholism. Of these 29.5 million sufferers, almost a million (894,000) were aged between 12 and 17. It’ll likely be some time before we know for sure whether the gene therapy can be rolled out in humans, but it’s an important first step in tackling this devastating disorder. Sign up for our free Indy100 weekly newsletter Have your say in our news democracy. Click the upvote icon at the top of the page to help raise this article through the indy100 rankings.

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